Advances in Dementia Research by D. Inzitari, G. Carlucci, Leonardo Pantoni MD. PhD (auth.),

By D. Inzitari, G. Carlucci, Leonardo Pantoni MD. PhD (auth.), Prof. Dr. K. Jellinger, Prof. Dr. R. Schmidt, Dr. M. Windisch (eds.)

Dementia is a tremendous ailment that turns into more and more universal with advancing age. regardless of contemporary development in neurobiology and molecular genetics, the aetiology and pathogenesis of so much dementia issues are nonetheless poorly understood, and early certain prognosis as a prerequisite of potent remedy has to be superior. the current quantity comprises the contributions of popular specialists within the box of neurodegeneration awarded on the foreign Symposium "Ageing and Dementia 1999”, September 24–26, 1999 at Graz. It focusses on genetics, epidemiology, new neuroimaging ideas, and the position of vascular, immunological and different mechanisms together with oxidative pressure and estrogens within the improvement of neurodegeneration and dementia. fresh advancements in prognosis and treatment of Alzheimer disorder and comparable issues are reviewed and destiny healing methods are mentioned. therefore, this quantity presents a well timed evaluate of latest advancements in dementia learn and remedy techniques of dementia disorders.

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The critical elements in the diagnosis ofVaD include the cognitive syndrome, vascular aetiologies and changes in the brain. , 1999a). , 2000). Clinical criteria for subcortical vascular dementia 25 The systematic heterogeneity in patients population derived by using current criteria has raised a need for updating of the sub-classification, which hold the potential to provide more homogenous subgroups. The main subtypes ofVaD include multi-infarct dementia or cortical VaD, strategic infarct dementia, and small vessel dementia or subcortical VaD (Erkinjuntti, 1987; Brun, 1994; Cummings, 1994; Wallin and Blennow, 1994).

Stroke 27: 1205-1210 Chabriat H, Joutel A, Vahedi K, Iba-Zizen M, Tournier-Lasserve E, Bousser M (1997) CADASIL (Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy). Rev Neurol (Paris) 153: 376-385 De Reuck J, Crevits L, De Coster W, Sieben G, Van der Eecken H (1980) Pathogenesis of Binswanger chronic progressive subcortical encephalopathy. Neurology 30: 920928 del Ser T, Bermejo F, Portera A, Arredondo JM, Bouras C, Constantinidis J (1990) Vascular dementia.

1999). They include non-CADASIL Binswanger-like syndromes without arterial hypertension, autosomal recessive leukoencephalopathy with alopecia and lumbago, and cerebroretinal vasculopathy. Other hereditary vascular conditions, unlinked to NOTCH 3 gene mutations and responsible for cerebral infarcts leading to dementia, have also been identified. , 1994). , 1995). White matter changes may be 34 D. Leys and F. , 1994), and probably contribute to the cognitive decline in post-stroke dementia (Pasquier and Leys, 1997).

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